Explain the table below:
| K + Channel |
Episodic ataxia type 1. Hyperinsulinemic hypoglycemia of infancy. Oncogenic potential. Benign familial neonatal convulsions. Hereditary hearing loss. Type II diabetes. Antenatal variant of Bartter syndrome. Andersen's syndrome. Total colour blindness. Periodic Paralysis. Long QT syndrome (type 1,2,5). Myokymia. |
| Na+ Channel | Liddle's syndrome. Hyperkaelemic Periodic Paralysis. Paramyotonia congenita. Generalized epilepsy with febrile seizures types 1 & 2. Long QT syndrome 3. Pseudohypoaldosteronism. Potassium-aggravated myotonia. Severe myoclonic epilepsy of infancy. Brugada syndrome. Isolated cardiac conduction disease. |
| Ca Channel | Episodic
ataxia type 2. Familial hemiplegic migraine. Spinocerebellar ataxia type 6. Hypokaelemic periodic paralysis type I. Malignant hyperthermia. Generalized epilepsy. Central core disease. Congenital night blindness. Expressed in advanced prostate cancer. Stationary night blindness. |
| Cl Channel | Myotonia congenita - (dominant or recessive). Dent's disease (proteinuria and hypercalciuria). Osteopetrosis. Bartter syndrome. Cystic Fibrosis. |
| Glycine receptor | Hyperekplexia
(stiff baby syndrome). Startle disease. |
| Acetylcholine receptor |
Congenital
myasthenia. Nocturnal frontal lobe epilepsy. |
| TRP Channels |
Polycystic
kidney disease. Mucolipidosis. Beckwith-Wiedemann-syndrome. Familial hypomagnesemia with hypocalcemia. |
| GABA-A receptor | Generalized epilepsy with febrile seizures. |
| Other |
Retinitis pigmentosa Gitelman Achromatopsia Darier disease. |